Practice Free EFM Certified - Electronic Fetal Monitoring Exam Questions Answers With Explanation

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

This tracing shows:

Baseline ~170–175 bpm ? fetal tachycardia

Minimal variability

No contractions of significance

Maternal treatment with magnesium sulfate, which typically decreases baseline and variability—not increase it

NCC and AWHONN physiology guidelines emphasize that fetal tachycardia is most commonly associated with maternal infection, including chorioamnionitis, especially in preterm labor.

Magnesium sulfate does not cause tachycardia; it generally causes:

? baseline

? variability

Thus, fetal tachycardia + minimal variability in a preterm patient strongly suggests maternal infection, requiring evaluation for chorioamnionitis.

Why the wrong answers are incorrect:

A. Acetaminophen ? used after confirming fever, not before evaluating the cause.

B. Discontinuing magnesium ? magnesium sulfate does not cause tachycardia; discontinuing it removes fetal neuroprotection.

Comprehensive and Detailed Explanation From Exact Extract NCC-Recommended Sources

The fetal heart rate is calculated from the interval between consecutive R waves in the fetal ECG. Shorter R-R intervals indicate more beats per unit of time, therefore resulting in a higher heart rate. AWHONN and Menihan both note that fetal ECG monitoring measures instantaneous rate based on R-R spacing, and “shorter intervals correspond to fetal tachycardia.”

Simpson & Creehan reinforce that fetal heart rate variability and baseline are derived from these R-R intervals, with shorter intervals consistently producing faster rates. Miller’s Pocket Guide describes the relationship simply: “Short R-R = faster rate; long R-R = slower rate.”

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

The tracing demonstrates:

Baseline: approx. 140 bpm

Variability: minimal-to-moderate (fluctuating but not consistently moderate)

Decelerations: shallow variable decelerations

Accelerations: not consistently present

According to NICHD/NCC definitions:

Category I requires ALL of the following:

Baseline 110–160

Moderate variability

No late or variable decelerations

Early decels and accelerations may be present

This tracing does not have consistently moderate variability and does have variable decelerations, so it is not Category I.

Category III requires ANY of the following:

Absent variability with recurrent late decels

Absent variability with recurrent variable decels

Absent variability with bradycardia

Sinusoidal pattern

This tracing does not show absent variability, bradycardia, or recurrent significant lates.

Category II includes:

Minimal variability

Absence of accelerations

Variable decelerations

Tracings not clearly Category I or III

This strip fits Category II exactly due to minimal variability + intermittent variable decelerations.

Thus, the correct classification is Category II.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

NICHD/NCC criteria:

Category I must have ALL of the following:

Baseline 110–160 bpm

Moderate variability

No late or variable decelerations

Early decelerations may be present or absent

Accelerations may be present or absent

Because this tracing has one variable deceleration, it fails Category I criterion (“no late or variable decelerations”).

Category III requires:

Absent variability with recurrent late decels, recurrent variables, or bradycardia, or

Sinusoidal pattern

Those findings are not present.

Therefore, any tracing that:

Has moderate variability and accelerations,

But includes a variable deceleration, and

Does not meet Category III criteria

…falls into the Category II (indeterminate) group.

Correct classification: B. Category II.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

NCC physiology content specifically includes maternal autoimmune influences on fetal cardiac conduction. Conditions such as maternal lupus (SLE) or Sjogren’s syndrome may produce anti-Ro/SSA and anti-La/SSB antibodies. These antibodies cross the placenta and damage fetal conduction tissue.

The primary site of injury is the fetal atrioventricular (AV) node, leading to:

First-, second-, or complete third-degree heart block

A slow, regular ventricular rate typically 50–70 bpm

Loss of beat-to-beat variability because ventricular myocardium does not display normal autonomic modulation

This mechanism is extensively described in AWHONN, NCC physiology materials, and maternal–fetal physiology texts.

Option A: Antibodies do not target fetal RBCs; that describes hemolytic disease of the newborn.

Option B: Targeting maternal WBCs is not fetal-specific.

The correct affected structure is the fetal AV node.

Therefore, the correct answer is C. The fetal atrioventricular node.

Comprehensive and Detailed Explanation (From NCC C-EFM–Referenced Sources)

According to NCC C-EFM content guidance and AWHONN Fetal Heart Monitoring Principles (2022), recurrent variable and late patterns in second stage with descent to +2/+3 station require consideration of expediting delivery, especially when maternal effort is prolonged and oxytocin augmentation is already present.

Menihan & Simpson emphasize that with prolonged second stage, continued pushing beyond 2–3 hours, and vertex at +3 station, the evidence-based next step is operative vaginal birth, provided prerequisites are met. Cesarean is not indicated when the fetal head is already low and deliverable vaginally.

AWHONN and Creasy & Resnik state that increasing oxytocin when facing fetal stress and prolonged second stage is contraindicated, because tachysystole worsens fetal oxygenation and increases risk of fetal compromise.

Exact Extract Concepts Referenced:

– “Expedited delivery is recommended when recurrent decelerations persist in second stage and the head is low enough for operative vaginal birth.” (AWHONN Principles)

– “Oxytocin should be reduced or discontinued in the presence of nonreassuring patterns.” (Simpson, Obstetric Interventions)

– “Operative vaginal delivery is appropriate with full dilation, engaged head, and prolonged second stage.” (Menihan, Simpson; Creasy & Resnik)

Comprehensive and Detailed Explanation From Exact Extract (No URLs or Links)

NCC-recommended physiologic texts (AWHONN, Menihan, Simpson, Creasy & Resnik) explain that variable decelerations are caused by umbilical cord compression. This process occurs in a three-step sequence, well known in fetal monitoring physiology:

Umbilical vein occlusion occurs first ? decreases fetal venous return ? brief fetal acceleration (a compensatory sympathetic response).

Umbilical artery occlusion follows ? increases fetal systemic vascular resistance ? variable deceleration as vagal stimulation lowers the fetal heart rate.

Release of compression ? post-deceleration acceleration may occur.

Thus, an acceleration immediately before a variable deceleration represents the initial compression of the umbilical vein, not a hypoxic response. This is a normal physiologic response to transient cord compression, often described in AWHONN and Menihan's physiologic explanation of “shoulders” around variable decelerations.

Oligohydramnios can contribute to cord compression but does not explain accelerations preceding the deceleration. A “hypoxic reflex” would not produce a pre-deceleration acceleration.

Therefore, the correct physiologic cause is:

Umbilical vein occlusion.

References (No URLs)

NCC C-EFM Candidate Guide 2025 – Physiology

AWHONN Fetal Heart Monitoring Principles

Menihan: Electronic Fetal Monitoring

Simpson & Creehan: Perinatal Nursing

Creasy & Resnik: Maternal–Fetal Medicine

Comprehensive and Detailed Explanation From Exact Extract NCC-Recommended Sources

The fetal heart rate tracing shows a normal baseline (120–150 bpm), moderate variability, and no decelerations, consistent with a Category I pattern. According to AWHONN’s Fetal Heart Monitoring Principles & Practices and NCC Perinatal Safety recommendations, a Category I tracing reliably indicates normal fetal acid-base status at the time of assessment and is considered reassuring.

Simpson & Creehan emphasize that in triage, management decisions depend on cervical status, contraction pattern, membrane status, and fetal well-being. With a cervix at 1 cm/50%/-2, intact membranes, and no regular labor pattern, she is not in active or latent labor requiring admission, provided fetal status is reassuring.

Menihan states that a normal tracing lasting two hours with moderate variability supports safe discharge when maternal and fetal assessments are normal. Creasy & Resnik confirm that reassuring fetal testing plus absence of labor is appropriate for outpatient management.

Comprehensive and Detailed Explanation From Exact Extract (NCC-Recommended Sources Only)

The fetal heart rate (FHR) tracing shown demonstrates a baseline approximately 135–145 bpm with fluctuations of 6–25 bpm, a hallmark of moderate variability. Moderate variability is defined in all NCC-endorsed resources as the normal amplitude range of 6–25 bpm around the fetal baseline.

According to the AWHONN Fetal Heart Monitoring Principles & Practices (2022–2024), moderate variability is considered the single most reliable indicator of adequate fetal oxygenation and intact neurologic pathways, specifically reflecting well-functioning sympathetic and parasympathetic interplay.

The NICHD/NCC standardized definitions included in the NCC C-EFM Candidate Guide state:

Minimal variability: amplitude range ? 5 bpm

Moderate variability: amplitude range 6–25 bpm

Marked variability: amplitude > 25 bpm

Sinusoidal pattern: smooth, undulating waveform, 3–5 cycles per minute, equal amplitude, absent beat-to-beat variability

The tracing provided does not show the repetitive, smooth, wave-like pattern of a sinusoidal rhythm; nor does it show flattening associated with minimal variability. Instead, it includes continuous beat-to-beat fluctuation within the moderate range, without periods of absent or minimal variability.

Menihan’s Electronic Fetal Monitoring (5th ed.) and Simpson & Creehan’s Perinatal Nursing (5th ed.) both emphasize that moderate variability is:

A reassuring feature

Indicative of adequate fetal CNS oxygenation

Expected in a reactive, well-oxygenated fetus

A key criterion for Category I classification

Additionally, Miller’s EFM Pocket Guide reiterates that variability between 6–25 bpm is considered the normal (moderate) fetal autonomic response and is not a sinusoidal pattern, which has a fixed amplitude and frequency.

Therefore, based on NCC-standard definitions and the observed amplitude, the correct interpretation is moderate variability.

References (No URLs):

AWHONN Fetal Heart Monitoring Principles & Practices; NCC C-EFM Candidate Guide 2025; Simpson & Creehan Perinatal Nursing; Menihan Electronic Fetal Monitoring; Miller’s Pocket Guide to Fetal Monitoring; Creasy & Resnik Maternal-Fetal Medicine.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

Amnioinfusion is considered an intrauterine resuscitative intervention used specifically for recurrent variable decelerations caused by cord compression. NCC, AWHONN, Miller, and Menihan consistently teach that variables occur when the umbilical cord becomes compressed, reducing fetal oxygenation. When oligohydramnios or decreased amniotic fluid volume is present, the cord is more vulnerable to compression.

Why amnioinfusion is used:

Amnioinfusion works by:

Increasing intraamniotic fluid volume

Reducing umbilical cord compression

Decreasing the frequency and severity of variable decelerations

This directly targets the pathophysiology behind recurrent variables.

Why the other options are incorrect:

A. Dilute thick meconium – NOT supported by NCC

Historically, amnioinfusion was studied for meconium dilution, but major organizations—including NCC-aligned sources—state that amnioinfusion is NOT recommended for the sole purpose of diluting meconium. It does not reduce meconium aspiration syndrome and is no longer indicated for that purpose.

B. Restore uterine blood flow – NOT accurate

Uterine blood flow is addressed through maternal positioning, fluid bolus, reducing uterine tachysystole, and minimizing vasoconstriction—not via amnioinfusion. Amnioinfusion does not physiologically affect uterine perfusion.

C. Treat oligohydramnios – CORRECT

Recurrent variables with thick meconium often occur in the setting of low fluid, which worsens cord compression.

NCC-recommended indications include:

Recurrent variable decelerations unresponsive to repositioning

Suspected or confirmed oligohydramnios

Thick meconium may be associated with low fluid, but the purpose of amnioinfusion is to alleviate cord compression by restoring fluid volume, not to dilute the meconium.

Thus, the correct answer is C. Treat oligohydramnios.

Comprehensive and Detailed Explanation From Exact Extract (NCC C-EFM sources: AWHONN, Miller’s Pocket Guide, Menihan, Simpson, Creasy & Resnik, 2025 Candidate Guide)

AWHONN and Menihan emphasize that most fetal dysrhythmias detected intrapartum are premature atrial contractions (PACs)—the most common benign rhythm variation. They typically appear as intermittent, irregular deflections on the fetal heart rate tracing without affecting variability or baseline.

Miller’s Pocket Guide to Fetal Monitoring states that PACs are usually transient, self-limiting, and require only observation unless accompanied by tachyarrhythmia or hemodynamic compromise. When variability is preserved and no repetitive pattern or sustained tachycardia occurs, no intrauterine resuscitation measures are indicated.

Simpson and Creehan describe that oxygen administration and fluid boluses are not recommended for benign dysrhythmias, as they do not improve fetal conduction patterns and may contribute to unnecessary interventions.

The NCC 2025 Candidate Guide specifies that correct management requires distinguishing benign arrhythmias from pathologic tachyarrhythmias, which would require escalation. In the absence of fetal compromise or maternal pathology, the appropriate action is continued observation.

Therefore, the correct management is to continue to observe.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

The lower uterine tracing shows a repetitive contraction pattern characterized by pairs of contractions occurring close together, followed by a longer interval. This is known as “uterine contraction coupling.”

Key features confirming coupling:

Two contractions occur back-to-back, separated by only a few seconds.

Then a longer rest period occurs before the next pair.

This pattern persists over several minutes.

FHR remains normal with moderate variability and no decelerations.

Coupling is a uterine activity pattern, not a fetal heart rate abnormality.

Why the other answers are incorrect

A. Category I tracing

While the FHR itself may appear reassuring, the question is explicitly about the pattern shown, which is (per NCC classification) a uterine pattern, not a category designation.

C. Prolonged acceleration

A prolonged acceleration would be a fetal heart rate increase ?15 bpm lasting ?2 minutes but <10 minutes.

No such FHR increase appears on the strip.

Thus, the correct interpretation is B. Coupling of contractions.

Comprehensive and Detailed Explanation From Exact Extract without any URL or Links

According to the NCC C-EFM 2025 Candidate Guide, Pattern Recognition and Intervention requires the candidate to classify fetal heart rate (FHR) patterns using the NICHD 2008 three-tier system, which NCC endorses across all recommended resources (AWHONN Fetal Heart Monitoring Principles and Practices, Menihan Electronic Fetal Monitoring, Simpson & Creasy, Miller’s Pocket Guide).

A Category II tracing is defined as “indeterminate” and includes any FHR pattern that is not Category I and not Category III. NCC references indicate that Category II may include:

Minimal or marked variability

Absence of accelerations after fetal stimulation

Recurrent variable decelerations with moderate variability

Prolonged decelerations lasting 2–10 minutes

Baseline tachycardia or bradycardia without absent variability

In the tracing provided:

The baseline FHR is approximately 135–145 bpm, within normal limits.

Moderate variability is not consistently present; variability is borderline minimal–moderate at times.

No significant accelerations are seen over the 25-minute evaluation period.

No recurrent late or prolonged decelerations are present.

There are occasional subtle variable-type dips, but not enough to meet criteria for Category III.

NCC-endorsed texts (such as AWHONN and Menihan) state that a tracing with minimal variability for less than 40 minutes and without recurrent decelerations is Category II, as it fails to meet the requirements for Category I (must have moderate variability and accelerations absent decelerations) and lacks the criteria for Category III (must have absent variability with recurrent late decels, recurrent variable decels, bradycardia, or sinusoidal pattern).

Therefore, this pattern is indeterminate, consistent with Category II, and requires continued surveillance and evaluation, which aligns with NCC-recommended clinical decision-making competencies.

Comprehensive and Detailed Explanation From Exact Extract (NCC-Referenced Sources)

According to AWHONN, Simpson, and NCC C-EFM physiologic competencies, uterine rupture commonly presents with:

Sudden prolonged deceleration

Recurrent variables

Fetal bradycardia

Possible loss of station, vaginal bleeding, maternal pain

AWHONN specifically lists:

“Prolonged deceleration is the most common initial fetal sign of uterine rupture.”

Absent variability can occur later, but it is not the most common initial pattern.

“Loss of uterine pressure” refers to loss of toco signal, not a fetal heart rate characteristic.

Therefore, NCC-validated interpretation: prolonged and variable decelerations.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

The tracing shows a clear relationship between uterine activity and fetal heart rate changes:

The uterine activity strip demonstrates very frequent contractions with little resting time between them, exceeding five contractions in 10 minutes, averaged over a 30-minute window.

NCC and NICHD define tachysystole as “more than 5 contractions in 10 minutes, averaged over 30 minutes, regardless of whether the labor is spontaneous or stimulated.”

As uterine activity intensifies and becomes excessively frequent, the fetal heart rate strip begins to show:

Progressive decrease in baseline

Recurrent decelerations with gradual onset and recovery

Reduced variability in the latter portion of the strip

This pattern is consistent with uteroplacental insufficiency caused by excessive uterine activity (tachysystole). NCC and AWHONN emphasize that tachysystole can result in decreased uterine blood flow and fetal oxygenation, leading to late or prolonged decelerations and eventual bradycardia if not corrected.

Why the other options are less likely:

A. Placental abruptionTypically associated with maternal symptoms (pain, vaginal bleeding, firm/boardlike uterus) and often a sustained increase in resting tone or a hypertonic contraction, not simply very frequent contractions. These maternal findings are not described in the vignette.

B. Rapid fetal descentUsually causes variable or early decelerations related to head compression, but the tocodynamometer would not necessarily show this degree of contraction frequency. The lower strip here clearly highlights excessive contractions as the primary problem.

Thus, the tracing’s FHR abnormalities are best explained by tachysystole, making C. Tachysystole the most appropriate answer.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

NCC and AWHONN emphasize unit-wide shared responsibility for:

Recognizing abnormal maternal or fetal findings

Calling for help

Triggering emergency responses (e.g., unit huddle, rapid response, safety pathways)

Safety culture requires:

Any staff member (RN, tech, provider) to initiate an alarm or escalate concern

No hierarchy delay

Rapid action when fetal compromise is suspected

Why the other answers are wrong:

A. Determined by unit leaders ? incorrect; safety is team-wide, not hierarchical.

C. Occur infrequently ? false; alarms must occur whenever needed, not limited.

Correct answer: B. Can be called by anyone.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

When bright red vaginal bleeding occurs at the moment of membrane rupture, accompanied by an acute, severe fetal heart rate deterioration, NCC sources emphasize considering conditions causing fetal hemorrhage rather than maternal instability.

The key features in this scenario:

Timing:Bleeding occurs immediately with spontaneous rupture of membranes—this is classic for vasa previa rupture, where fetal vessels traverse membranes and are torn when the membranes rupture.

Bleeding characteristics:Bleeding is acute, bright red, and sudden.In vasa previa, the blood observed vaginally is fetal blood, not maternal blood.

Uterine exam:The uterus is soft and nontender, which strongly argues against abruptio placenta, where the uterus is typically firm, rigid, or painful.

Maternal vital signs:Maternal blood pressure and pulse are normal, indicating no maternal hypovolemia.In placental abruption or placenta previa with significant maternal bleeding, maternal vitals are often abnormal.Here, the mother is stable, meaning the blood is not maternal—supporting fetal vessel rupture.

Fetal heart rate pattern:

Initial tachycardia, followed by

Bradycardia with minimal variabilitySuch a pattern is consistent with acute fetal blood loss, which rapidly leads to fetal hypovolemia and hypoxia.

Differential based on NCC-aligned physiology:

A. Abruptio placenta – NOT supported

Typically presents with:

Painful bleeding

Firm, tender uterus

Maternal tachycardia

Uterine irritabilityNone of these are present.

B. Placenta previa – NOT supported

Classically painless bright red bleeding before or early in labor, not triggered by membrane rupture.

Fetal compromise is less sudden unless maternal shock occurs, which is not the case here.

C. Ruptured vasa previa – CORRECT

Defined by:

Painless, sudden bright red bleeding at ROM

Normal maternal vital signs

Rapid fetal deterioration (tachycardia ? bradycardia ? minimal variability)

Soft, nontender uterusThis fits the scenario exactly.

Therefore, the most likely cause is ruptured vasa previa, a recognized obstetric emergency described across AWHONN, NCC C-EFM references, and maternal–fetal physiology texts such as Menihan and Creasy & Resnik.

Comprehensive and Detailed Explanation From Exact Extract Sources:

The tracing demonstrates an abrupt-onset, sharp, V-shaped deceleration, occurring simultaneously with or slightly after a contraction—classic for variable decelerations, which are caused by umbilical cord compression.

According to AWHONN Fetal Heart Monitoring Principles & Practices, variable decelerations are defined by:

“Abrupt decreases in FHR below baseline of at least 15 bpm, lasting at least 15 seconds and less than 2 minutes.”

“Most commonly associated with umbilical cord compression, whether transient or recurrent.”

Physiology reference (Simpson & Miller, Pocket Guide):

Compression of the umbilical vein causes a brief acceleration.

Compression of the umbilical arteries triggers a vagal response, producing a rapid deceleration.

This creates the characteristic sharp ‘V’, ‘U’, or ‘W’ shape on the monitor.

Placental insufficiency (Choice B) produces late decelerations, which are gradual, not abrupt.

Fetal head compression (Choice A) produces early decelerations, which mirror contractions and have a gradual pattern.

Thus, the tracing is most consistent with variable decelerations caused by umbilical cord compression.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

Tachysystole = >5 contractions in 10 minutes averaged over 30 minutes (NICHD).

However, NCC and AWHONN intervention guidelines state:

If tachysystole appears in one or two consecutive 10-minute segments, especially with Category II or III patterns, intervention must begin immediately.

Intervention includes:

Stopping/reducing oxytocin

Maternal repositioning

IV bolus

Tocolysis if needed

Why the wrong answers are wrong:

A. Waiting 30 minutes delays necessary fetal resuscitation.

C. Stage of labor does not determine when to intervene.

Correct answer: B. If tachysystole is seen for one or two 10-minute segments

Comprehensive and Detailed Explanation From NCC-Aligned Emergency Fetal Response Principles:

Following an eclamptic seizure:

Maternal hypoxia, apnea, and intense sympathetic discharge occur

Uteroplacental perfusion drops

Fetus experiences acute hypoxemia

The expected fetal heart rate response is a prolonged bradycardia

This is well-described in NCC and AWHONN emergency physiology:

“Post-seizure fetal bradycardia is common and often resolves within 5–10 minutes as maternal oxygenation stabilizes.”

Why other answers are incorrect:

B. Sinusoidal pattern — Rare and usually indicates fetal anemia, not post-seizure status.

C. Variable decelerations — Associated with cord compression, not seizures.

Correct answer: A. Bradycardia

Comprehensive and Detailed Explanation From Exact Extract NCC-Recommended Sources

Fetal tachycardia is typically caused by maternal fever, dehydration, hypoxia, medications, infection, or fetal stress. AWHONN and Simpson & Creehan emphasize that management focuses on improving oxygen delivery across the placenta, which is governed by uteroplacental perfusion.

Menihan’s EFM text states that “interventions for fetal tachycardia must address oxygen transfer by optimizing uteroplacental blood flow,” including hydration, reducing uterine activity, maternal repositioning, and treating maternal fever.

Increasing maternal circulation alone is insufficient unless it improves placental blood flow. Enhancing fetal sympathetic tone is not a clinical goal and would worsen tachycardia.

Creasy & Resnik highlight that fetal heart rate abnormalities resolve when uteroplacental perfusion is restored, confirming this as the primary target of intervention.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

NCC’s Professional Issues domain includes EMTALA obligations for pregnant patients. EMTALA requires that ANY individual who presents to a hospital emergency department—regardless of insurance status—must receive:

A Medical Screening Examination (MSE)

Stabilization of any identified emergency medical condition (including labor)

No transfer unless the patient requests it or the hospital cannot provide necessary stabilizing care

This patient reports contractions at 36 weeks, which qualifies as a potential emergency medical condition until ruled out by the medical screening exam.

Correct obligations per EMTALA:

She must NOT be transferred solely due to lack of insurance (option C).

She does NOT need to be admitted unless labor is confirmed (option A).

She must receive a medical screening examination and stabilization (option B).

Thus, the correct answer is B. Stabilized and receive a medical screening examination.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

When internal monitoring continues to record artifact despite:

Changing the scalp electrode

Ensuring correct attachment

Checking cable connections

Confirming maternal movement is not the cause

NCC requires confirmation of fetal well-being using another modality.

The correct next step is direct auscultation with Doppler or fetoscope.

Why other answers are incorrect:

Oxygen is not indicated for equipment malfunction.

Repositioning does not resolve internal FHR artifact.

Thus, Auscultate the fetal heart rate is the appropriate next step.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

Accurate classification of decelerations requires evaluating their shape, onset, nadir, recovery, relationship to contractions, and variability characteristics. NCC uses the NICHD standardized definitions, reinforced across AWHONN, Miller’s Pocket Guide, Menihan, Simpson, and Creasy & Resnik.

Key features in this tracing:

Abrupt onsetThe FHR drops rapidly from baseline to nadir in less than 30 seconds—this is the defining hallmark of a variable deceleration per NICHD.

Sharp V-shape and deep amplitudeThe tracing shows steep descents and ascents, characteristic of cord compression–type variable decelerations.

Inconsistent timing with contractionsThe decelerations do not begin at the start of contractions (as early decelerations would) and do not consistently begin after the peak of contractions (as late decelerations would). Variable decelerations can occur before, during, or after a contraction—exactly what is demonstrated here.

Rapid return to baselineAnother core feature of variable decelerations in NICHD/NCC definitions.

No uniform contraction relationshipEarly decelerations are symmetrical and mirror contractions. Late decelerations begin after the peak of the contraction. This strip does not match either pattern.

Differentiation per NCC-aligned definitions:

Early Decelerations:Gradual onset (>30 sec), nadir mirrors contraction peak, shallow, uniform.Not present.

Late Decelerations:Gradual descent, nadir after contraction peak, smooth shape.Not present.

Variable Decelerations:Abrupt onset (<30 sec), variable timing, sharp V-shape, rapid recovery, often with shoulders.Exactly matches the tracing.

Therefore, according to NICHD/NCC criteria, the decelerations shown are variable decelerations.

Comprehensive and Detailed Explanation From Exact Extract–Based NCC C-EFM References:

During amnioinfusion, NCC emphasizes monitoring for uterine overdistention, which can lead to uterine hypertonus, uterine rupture, or placental separation. The primary way to evaluate overdistention is by measuring baseline uterine pressure via IUPC.

Rising resting tone (>20–25 mmHg) indicates accumulating fluid and risk.

Stopping the infusion intermittently allows recalibration and assessment of uterine baseline pressure.

Contraction pattern (option B) is important but not the primary safety parameter.

Pain (option C) is nonspecific and not a reliable indicator of uterine overdistention.

Thus, the infusion is stopped to assess baseline uterine pressure.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

NICHD definitions:

A variable deceleration is identified by:

Abrupt onset(drop from baseline to nadir in <30 seconds)

Depth ?15 bpm

Duration ?15 seconds and <2 minutes

Variable timing relative to contractions

Variable shape (sharp drop, jagged descents, rapid recovery)

The scenario describes:

Abrupt drop from 125 ? 80 bpm (rapid onset)

Lasting 90 seconds (still <2 minutes)

Gradual return but still within variable range

Occurring during a contraction

Depth >15 bpm

This meets ALL criteria for a variable deceleration.

Why the other options are wrong:

A. Early deceleration

Requires gradual onset (>30 seconds).

Mirrors contraction shape.

Caused by head compression.

This decel is abrupt, so NOT early.

B. Prolonged deceleration

Requires ?2 minutes and <10 minutes.

This decel lasts 90 seconds, which is below the threshold.

Correct classification: Variable deceleration.

Comprehensive and Detailed Explanation From Exact Extract Without Links:

NCC-recommended references (Simpson, AWHONN FHM, Creasy & Resnik) note that baseline increases within the normal range (110–160 bpm) accompanied by moderate variability are typically benign. Mild physiologic causes—maternal activity, fetal stimulation, or normal sympathetic activation—may transiently raise baseline FHR.

AWHONN stresses that intervention is required only when tachycardia exceeds 160 bpm or when variability is minimal/absent or accompanied by recurrent decelerations.

Here, the baseline increase to 150 bpm remains within normal limits and is paired with moderate variability, which the NCC recognizes as the strongest indicator of adequate fetal oxygenation.

Therefore, evaluation is complete, and continued observation is the appropriate course.

Comprehensive and Detailed Explanation From Exact Extract Sources:

In fetal physiology, the highest oxygen saturation exists in the umbilical vein, which then flows through the ductus venosus before entering the right atrium.

According to Creasy & Resnik Maternal-Fetal Medicine, and AWHONN physiologic foundations:

The umbilical vein carries oxygen-rich blood from the placenta (approx. 80% saturation).

Most of this blood bypasses the liver via the ductus venosus, which therefore contains the most highly oxygenated blood within the fetal circulatory system.

By contrast:

The descending aorta contains mixed blood with significantly lower oxygen content due to mixing after passage through the ductus arteriosus.

The pulmonary arteries in the fetus carry predominantly deoxygenated blood, since fetal lungs are fluid-filled and have high pulmonary vascular resistance.

Thus, the structure containing the highest fetal oxygen concentration is the ductus venosus.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

Vagal stimulation is part of the parasympathetic nervous system, which causes:

Slowing of the fetal heart rate (FHR)

Rapid but temporary changes in HR

Seen with head compression, scalp stimulation, or fetal movement

NICHD/NCC physiology explains:

Vagus nerve activation ? acetylcholine release ? slowed SA node firing ? decrease in FHR

This mechanism is responsible for early decelerations during labor due to head compression.

Why the incorrect answers are wrong:

B. Increased cardiac contractility ? sympathetic effect, not vagal.

C. Increased fetal blood pressure ? also a sympathetic effect.

Correct answer: A. Decreased fetal heart rate

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

Category III criteria include:

Absent variability with recurrent variable decelerations

Absent variability with recurrent lates

Absent variability with bradycardia

Sinusoidal pattern

Thus, recurrent variables become Category III when accompanied by absent variability, indicating fetal decompensation.

Why the other answers are wrong:

B. Late decelerations ? Category III only if combined with absent variability.

C. Tachysystole ? Contraction pattern, not a FHR characteristic.

Correct answer: Absent variability.

Comprehensive and Detailed Explanation From NCC-Aligned Sources:

According to NCC, AWHONN, and evidence-based documentation standards, clinicians must document:

Baseline

Variability

Accelerations

Decelerations (type, depth, duration, timing)

Uterine activity

This fulfills the NICHD 3-tier system and legal documentation expectations.

Why the incorrect answers are wrong:

B. "Normal/abnormal" ? vague, not an acceptable documentation standard.

C. Category alone ? insufficient; categories must be supported by the components.

Comprehensive and Detailed Explanation From Exact Extract (NCC-Referenced Sources)

According to the NCC C-EFM Exam Outline and AWHONN Fetal Heart Monitoring (5th & 6th ed.), recurrent variable decelerations with progressive reduction in variability reflect worsening fetal hypoxia, especially when coupled with prolonged second stage and arrest of descent.

AWHONN and Menihan both state that:

“Minimal variability with recurrent decelerations indicates inability of the fetus to maintain adequate oxygenation.”

“Failure of descent in second stage with non-reassuring patterns requires operative delivery.”

Creasy & Resnik emphasize that operative vaginal birth requires:

(1) fetal head at +2 station or below,

(2) favorable position,

(3) reassuring fetal status.

Here, the fetus is OP, descent has arrested, and FHR is non-reassuring. This contraindicates vacuum extraction.

Therefore, the appropriate management under NCC competencies is cesarean birth.